Proinflammatory HIV-1 Pathogenesis and Precarcinogenesis

doi:ijmmas.2005.105.111

eISSN: 1994-4624 / ISSN: 1813-176x

International Journal of Molecular Medicine and Advance Sciences

2005, Volume 1, Issue 2 : 105-111 doi: ijmmas.2005.105.111

Research Article

Proinflammatory HIV-1 Pathogenesis and Precarcinogenesis

DOI : ijmmas.2005.105.111

Published

June 30, 2005

Abstract

HIV-1 infection assumes the features of a systemic process affecting cellular cycling events and of inflammatory and neoplastic amplification. It would indeed appear that HIV-1 induces pathways of progression that are specifically transforming in terms of such amplification and self-amplification of cellular and microenvironmental events. The central nervous system in particular would constitute a manifestation of organ integrity that expresses the consequences of amplified transformation of pathobiologic lesions. Neoplasia of the lymphoreticular system would represent a characterization of the nature of such events as further projected by the HIV-1 infection evolving as AIDS dementia and Primary Lymphoma of the CNS and as Progressive Multifocal Leukoencephalopathy or opportunistic infection in general.

Keywords

HIV-I

Precarcinogenesis

Systemic process

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